How Does It Work:



Mitochondria perform diverse yet interconnected functions, producing ATP and many biosynthetic intermediates while also contributing to cellular stress responses such as autophagy and apoptosis.


Mitochondria form a dynamic, interconnected network that is intimately integrated with other cellular compartments. In addition, mitochondrial functions extend beyond the boundaries of the cell and influence an organism's physiology by regulating communication between cells and tissues.


It is therefore not surprising that mitochondrial dysfunction has emerged as a key factor in a myriad of diseases, including neurodegenerative and metabolic disorders. 

The entire body at its core is comprised of cells. 37. 2 trillion cells to be exact. At the centre of most of those cells is the control centre – or the mitochondria. It takes a bit of cooperation amongst cellular processes to keep us well and alive, and all that science-y stuff is actually pretty cool.

It’s also incredible to consider the link between things like the gut microbiome and mitochondria, and how that relationship impacts our overall health. The best part of all is how much we can make a positive impact on those processes through the healthy choices we make.

In essence, mitochondria have much to do with our bodily functions and disease. Mitochondria respond to nutrition – think of them as the “digestive system” of the cells.

Mitochondria are designed with specialised functions to efficiently break down carbohydrates and essential fatty acids, turning them into energy that is available to the cells for use or adenosine triphosphate (ATP). This powers the cells; it is the gas in our fuel tanks on the most basic level.

The mitochondria are intelligent, and it knows how to allocate energy after breaking it down! It only makes sense that the foods we choose to eat play a crucial role in the optimisation of mitochondrial functioning. The “mysteries” of mitochondria are uncovered in their interaction with other cells and overall cellular functioning. Therefore, determining the way this facet of health impacts our well-being and the way we can make an impact on that is highly specific and complex.


  • Heart disease. Essentially, the mitochondria don’t receive enough oxygen as early onset heart failure compromises the availability of oxygen and triggers a stress signal to the body to tell it so. Delivering antioxidants to the cells can potentially lessen dysfunction and prevent heart failure.

  • Cancer. DNA damage – identified within cells – is an early marker of cancer. Mutated mtDNA cells are a telling sign of malignant tumours and their growth. Above all, cancer is a cellular disease at its core, so the health of our mitochondria plays a role in both prevention and therapeutic treatment. By optimising mitochondrial metabolism, we can do our best to slim down the chance of cancer.

  • Mitochondrial disease. This is a very broad spectrum of disease, and there are literally hundreds of mitochondrial diseases. These manifest as a result of mutations in mtDNA. Finally, you should know that different mutations can cause the same disease, so it’s not always easy to trace the trouble to the source.

  • Fatigue. While fatigue isn’t quite as life-threatening as cancer, it can be a painful and persistent problem with seemingly no solution. Visiting the doctor for prolonged fatigue is not going to be a clear diagnosis. Poor mitochondria function is to be blamed. Prolonged use of certain medications, antibiotic use, and other factors can diminish ATP production and leave our cells without the energy they need to function at peak performance.


Unfortunately, at the Doctor's office, they don't ask you "How is your mitochondrial health? Usual Conversation with The Doctor goes more or less like this:

"Dear GP, how to make my heart age slower?"

GP: "You should ask a cardiologist"

"Dear cardiologist, how to make my intestine age slower?"

Cardiologist: "You should ask a gastrologist"

"Dear gastrologist, how to make my whole body age slower?"

Gastrologist: "You should ask your GP"